荊芥薄荷的問題，透過圖書和論文來找解法和答案更準確安心。 我們找到下列包括賽程、直播線上看和比分戰績懶人包

調香手記：55種天然香料萃取實錄

為了解決荊芥薄荷的問題，作者蔡錦文 這樣論述：

　　香水達人居家萃取天然香料，歷經七年的完整手記。 　　柑橘、茉莉、咖啡、薄荷、木香、睡蓮…… 　　四種香料萃取法˙55種天然香料， 　　無需昂貴設備就能提取原精，調配香水， 　　更可廣泛運用在芳療、手工皂、保養品等香氛創作上， 　　打造專屬自己、獨一無二的自然香氣。 　　香，為生活帶來美好感受， 　　人們也順應著喜愛香氣的本能與天性， 　　將香氣具體化成香水、香膏、精油……， 　　應用於日常中，豐富與潤澤我們的生活。 　　不同於化學合成的人工香精， 　　天然香料製成的香水，蘊含動植物的鮮活氣息， 　　有著人工香水無法比擬的香調， 　　且氣味無法被如實複製，而是隨著使用者的身體一起轉

化， 　　呈現出來的香味獨特，是極具生命力的動態香水。 　　作者蔡錦文本是一個對香料、香水一無所知的野鳥生態畫家， 　　因為一瓶Mystra（Aesop）香水的神祕薰香， 　　從此愛上天然香料簡單富足、雋永有味的本質， 　　終日投入研究香料萃取，研製天然香水。 　　七年來，閱讀無數中外文獻，不斷嘗試實驗， 　　累積豐富的萃香經驗，首度集結大公開， 　　獻給喜歡萃香、調香、閱讀植物氣味的你。 　　■300多張超清楚圖解，跟著做，超實用！ 　　居家就可進行的合香、浸泡、擠壓、脂吸四種萃香方法，配備簡單調香工具、調香基本知識，解說天然香料萃取方法與運用。全書詳細圖解，初學者也能輕鬆上手。萃取出的

天然香料，除了用來調配香水，也可運用在芳療、製作手工皂、按摩油、面霜、香磚等香氛保養品中。 　　■5大類、55種天然香料萃取實錄，追求天然香料、發掘本土香料的最佳指南 　　宛如現代葛奴乙的錦文，從最簡單親民的柑橘、丁香、迷迭香開始，進而深入生活環境中尋找值得開發的香料，如咖啡、花椒、鳳梨等。近年，更投入從山棕、山黃梔、台灣香檬等本土植物與中藥材中萃取香料，提煉出與眾不同的獨特香氣。 　　■七年淬鍊出的調香心得大公開，做自己的調香師！ 　　深入分析每一種香料萃取時的關鍵要領與調香建議，創造專屬自己，獨具風格與手感的天然香水，盡情享受、操使氣味，體驗調香樂趣！ 本書特色 　　1.圖解四種香

料萃取法，入門者必備不敗祕笈。 　　2.細說55種天然香料的特色與萃取法，提供調香建議與技巧，為自己打造獨一無二的香氣。 　　3.獨家收錄本土芳香植物與芳香中草藥的應用。 　　4.對於氣味的專業分析與生動描述，開啟嗅覺記憶的奇妙旅行。 　　5.介紹天然香水歷史，完整記錄55種天然香料萃取實錄，兼具人文與實用價值。  

荊芥薄荷進入發燒排行的影片

中草藥粗萃物抑制腸病毒71型之研究

為了解決荊芥薄荷的問題，作者陳信光 這樣論述：

Recommendation Letter from the Thesis AdvisorThesis/Dissertation Oral Defense Committee CertificationAcknowledgments……………………………………………………iiiChinese Abstract……………………………………………………ivEnglish Abstract………………………………………………………viTable of Contents………………………………………………………viiiList of Figures……………………………………………………………xii

iList of tables………………………………………………………………xviChapter 1. Introduction…………………………………………………1Section 1.1 Human enterovirus………………………………………11.1.1 Classification………………………………………………………11.1.2 Epidemiology of EV71……………………………………………21.1.3 Clinical manifestations of EV71…………………………………21.1.4 EV71 viral particle……………………

…………………………31.1.5 EV71 life cycle……………………………………………………41.1.6 EV71 morphogenesis……………………………………………61.1.7 Reactive oxygen species accumulation and EV71 pathogenesis…61.1.8 Prophylaxis and treatment of EV71 infections…………………7Section 1.2 Chinese herbal medicine and medicinal herb………………81.2.1 The opinion of

traditional Chinese medicine about HFMD……81.2.2 Ma Xing Shi Gan Tang…………………………………………81.2.3 Ko Keng Tang……………………………………………………91.2.4 Chuan Xiong Cha Tiao San………………………………………91.2.5 Schizonepeta tenuifolia Briq. ……………………………………101.2.6 Mentha haplocalyx Briq. ………………………………………101.2.7 Melissa officinalis……………

…………………………………11Section1.3 Specific aims………………………………………………12Chapter 2. Materials and methods………………………………………132.1 Chemicals………………………………………………………132.2 Traditional Chinese medicine and herbal extract preparation…142.3 Cells and viruses…………………………………………………152.4 Viral plaque assays and plaque reduction ass

ays…………………172.5 Cell viability assay………………………………………………182.6 Determine related copy number of EV71…………………………182.7 Cytopathic effect (CPE) assay……………………………………192.8 Viral progeny generation assay……………………………………192.9 SDS-PAGE and western blot analysis……………………………202.10 LC-MS method…………………………………………………212.1

1 Proton nuclear magnetic resonance (1H-NMR)…………………222.12 Time-of-addition assay……………………………………………222.13 Infectivity reduction assay…………………………………………232.14 Analyzed the switch between cap- and IRES-dependent translation……………………………………………………………242.15 Heterogeneous nuclear ribonucleoprotein A1 translocat

ion assay…………………………………………………………………252.16 Treatment of cells with p38 MAPK inhibitor, SB202190……262.17 Ferric reducing/antioxidant power (FRAP) assay……………262.18 Detection of cellular reactive oxygen species (ROS)……………272.19 The induction of p38 MAPK phosphorylation by hydrogen peroxide………………………………………

…………………………282.20 Ethic statement……………………………………………………282.21 Animal study………………………………………………………282.22 Statistical analysis………………………………………………29Chapter 3. Results………………………………………………………30Section 3.1 Pre-screening of anti-enterovirus 71 from antipyretic traditional Chinese medicinal formulas………………………………303.

1.1 Water extracts of MXSGT, KKT and CXCTS show anti-EV71 activity………………………………………………………………303.1.2 CXCTS inhibits EV71 infection………………………………313.1.3 CXCTS suppresses EV71 infection during virus attachment and post-attachment stages………………………………………………323.1.4 CXCTS inhibits EV71-induced shutoff of cap-de

pendent translation and enhance of IRES-dependent translation………………32Section 3.2 Antiviral activities of Schizonepeta tenuifolia Briq. against enterovirus 71 in vitro and in vivo……………………………………353.2.1 Water extract of Schizonepeta tenuifolia Briq. (STE) shows anti-EV71 activity……………………………………………………353

.2.2 STE inhibits EV71 infection at both absorption and post-absorption phases………………………………………………363.2.3 STE retarded the EV71-induced switch between cap-dependent and IRES-dependent translation in cells………………………………373.2.4 STE decreases EV71-induced phosphorylation of p38 kinase…393.2.5 The p38 signa

ling pathway is important for EV71 infection…403.2.6 STE suppresses EV71-induced phosphorylation of p38 MAPK through antioxidant mechanism………………………………………413.2.7 EV71 infection activates the phosphorylation of epidermal growth factor receptor substrate 15 (EPS15) at Ser796……………422.3.8 STE treatment e

nhances the survival rate and alleviates clinical symptoms in EV71-infected mice……………………………………43Section 3.3 Anti-enterovirus 71 activities of Mentha haplocalyx Briq. in vitro………………………………………………………………433.3.1 MHB displays anti-EV71 activity without cytotoxicity………433.3.2 MHB suppresses EV71 replication

……………………………443.3.3 MHB represses EV71 infection at both attachment and post-attachment phases………………………………………………453.3.4 MHB prevents EV71-induced imbalance of cap- and IRES- dependent translation…………………………………………………46Section 3.4 Anti-enterovirus 71 activities of Melissa officinalis extract and its bio

logically active constituent rosmarinic acid……………483.4.1 Introduction…………………………………………………483.4.2 Methanolic extract of MO (MOM) has antiviral activity……493.4.3 Identification of antiviral constituents in MOM……………503.4.4 RA inhibits EV71 replication………………………………513.4.5 RA inhibits EV71 infection at att

achment and post-attachment stages……………………………………………………………523.4.6 RA represses EV71-indcued switch between cap-dependent and IRES-dependent translation………………………………………533.4.7 RA repressed EV71-induced the hyper-phosphorylation of p38 mitogen-activated protein kinase (MAPK)……………………………553.4.8 RA suppres

sed EV71-induced accumulation of ROS which active p38 signaling…………………………………………………………563.4.9 EV71 infection stimulates phosphorylation of epidermal growth factor receptor substrate 15 (EPS15) via p38 signaling……………573.4.10 RA protects mice from EV71 infection………………………57Chapter 4. Discussion and conc

lusion…………………………………59References…………………………………………………………………71Appendix…………………………………………………………………99List of figuresFigure 1. Anti-EV71 activity of antipyretic Chinese medicine formulas…………………………………………………………………99Figure 2. CXCTS suppresses EV71 infection…………………………101Figure 3. CXCTS represses EV71 infection a

t viral attachment and post-attachment phase…………………………………………………103Figure 4. CXCTS inhibits EV71-induced shutoff of host protein synthesis and turn-on of IRES-dependent translation………………105Figure 5. STE inhibits EV71-induced plaque formation in RD and Vero cells without cytotoxicity………………………………………………

107Figure 6. STE inhibits EV71 replication…………………………109Figure 7. STE suppresses EV71 infection at attachment and post- attachment stages……………………………………………………112Figure 8. STE suppressed the EV71-induced shutoff of cap-dependent translation and initiation of IRES-dependent translation in host cells……11

5Figure 9. STE suppressed EV71-induced phosphorylation of p38 MAPK rather than that of Erk……………………………………………117Figure 10. The p38 signaling pathway is needed for EV71 replication………………………………………………………………119Figure 11. STE alleviates EV71-induced oxidative stress and hydrogen peroxide stimulated p38 pho

sphorylation………………………………121Figure 12. STE repressed EV71-induced phosphorylation of EPS15 at Ser796…………………………………………………………………123Figure 13. STE treatment protects mice from EV71 infection………124Figure 14. Anti-EV71 activity of MHB………………………………126Figure 15. MHB suppresses EV71 infection…………………………128Figur

e 16. MHB represses EV71 infection at viral attachment and post-attachment stages…………………………………………………131Figure 17. MHB suppresses EV71-induced shutdown of cap-dependent translation and EV71-stimulated enhance of IRES-dependent translation……………………………………………………133Figure 18. Anti-EV71 activity of methano

lic extract of MO (MOM) was verified……………………………………………………………………136Figure 19. Inhibition of enterovirus 71 (EV71)-induced cytopathic effect (CPE) by MOM……………………………………………………138Figure 20. Antiviral constituents in MOM were further purified…140Figure 21. Antiviral ingredients in MOM were analyzed by LC-M

S and LC-MS/MS……………………………………………………………142Figure 22. The MOMW1-1-1 was purified through column chromatography and it was identified as rosmarinic acid by proton nuclear magnetic resonance (1H-NMR)…………………………………144Figure 23. RA suppressed EV71-induced plaque formation showed low cytotoxic to RD and Vero

cells…………………………………………146Figure 24. RA inhibited EV71 replication……………………………148Figure 25. RA inhibited EV71 infection during viral absorption and post-absorption phases…………………………………………………151Figure 26. RA disturbs the switch between cap-dependent translation and IRES-dependent translation induced by

EV71 infection in host cells……………………………………………………………………154Figure 27. RA inhibits EV71-induced hyper-phosphorylation of p38 which is essential for EV71 replication and regulates the translocation of hnRNP A1……………………………………………………157Figure 28. RA suppressed EV71-induced ROS and hydrogen peroxide induced

p38 activation……………………………………………159Figure 29. RA reduces EV71-induced phosphorylation of EPS15 at Ser796…………………………………………………………………161Figure 30. RA protected mice from EV71 infection……………162Figure 31. RA is one active constitute in STE and MHB…………164Figure 32. A proposed model of drugs against EV71 inf

ection is shown in the scheme………………………………………………………165List of tablesTable 1. Antibodies used in this study……………………………167Table 2. Primers used in RT-PCR…………………………………168Table 3. Constituents known to be present in Schizonepeta tenuifolia Briq………………………………………………………………169Table 4. Compounds known to be exi

sting in Mentha haplocalyx Briq.………………………………………………………………171Table 5. Compounds known to be existing in Melissa officinalis…173

冉雪峰本草講義

為了解決荊芥薄荷的問題，作者冉雪峰 這樣論述：

內容包括卷一；補益類；人參；黃芪；白術；茯苓；當歸；芍藥；丹參；枸杞；龍眼肉；酸棗仁；沙參；麥門冬；百合；薯蕷；薏苡仁；何首烏；山茱萸；地黃；玄參；肉蓯蓉；補骨脂；巴戟天；淫羊藿；菟絲子；覆盆子；茺蔚子；人乳；膃肭臍；鹿茸；阿膠；雞子黃；大棗；甘草等。冉雪峰(1879年11月一1963年1月)，四川省巫山縣黛溪鎮人(今重慶市巫山縣大溪鄉)。先生六世醫傳，為全國著名中醫、傷寒臨床家。早在20世紀30年代，醫壇已有「南冉(雪峰)北張(錫純)」之譽。冉雪峰曾當選湖北省中西醫會第一屆正會長，創辦湖北私立中醫專門學校。1955年11月奉調入京，任中醫研究院學術委員會副主任委員兼高干外賓治療室主任。

卷一補益類人參黃芪白術茯苓當歸芍藥丹參枸杞龍眼肉酸棗仁沙參麥門冬百合薯蕷薏苡仁何首烏山茱萸地黃玄參肉蓯蓉補骨脂巴戟天淫羊藿菟絲子覆盆子茺蔚子人乳膃肭臍鹿茸阿膠雞子黃大棗甘草卷二解表類麻黃桂枝葛根柴胡荊芥薄荷紫蘇防風羌活秦艽升麻前胡水萍青蒿香薷西河柳木賊草白薇桑葉菊花蔥白通便類大黃硝石才卜石肖火麻仁郁李仁巴豆蓖麻子蘆薈豬膽汁番瀉葉柿霜蜂蜜卷三利尿類澤瀉豬苓滑石木通燈心草車前子知母蘆根茵陳蒿蒲黃蝸牛溫寒類附子烏頭草烏頭肉桂干姜高良姜吳茱萸蜀椒胡荽陽起石石硫黃除熱類犀角羚羊角黃連黃柏黃芩梔子連翹龍膽草青黛石膏淡竹葉……卷四卷五卷六卷七卷八

銀翹散對急性肺部發炎老鼠模式的保護作用及免疫調節之研究

為了解決荊芥薄荷的問題，作者葉家舟 這樣論述：

急性肺損傷(Acute lung injury; ALI)是造成患者死亡一個重要的成因。如何抗發炎的治療或是改善ALI患者的預後是重要的課題，己有許多文獻指出中醫藥在對抗肺部急性發炎有其成效，而銀翹散便是其中對抗肺部急性發炎重要的方劑，但其抗發炎的機制仍然不清楚。為了研究銀翹散對抗肺部急性發炎的機制，我們以氣管投與LPS誘發老鼠肺部急性發炎，同時餵食銀翹散，結果發表不論在肺部發炎的病理組織上、中性白血球的浸潤數量、肺部水腫指標及一氧化氮分泌的量上，皆可見到銀翹散有減緩肺部發炎的成效。我們以ELISA 及 RT-PCR 來偵測各種發炎相關的細胞激素及趨化因子，如TNFα、IL-1β、 IL-6

、KC、MCP-1及MIP-2，銀翹散皆有壓制的作用，同時我們也發現銀翹散可誘發大量的抗發炎細胞激素IL-10的分泌。我們利用鼠巨噬細胞RAW 264.7針對TNFα及IL-10 進行體外研究。銀翹散、銀花、連翹及牛蒡對LPS誘發產生TNF-α有明顯壓抑作用，荊芥、薄荷及淡竹葉少許壓制作用。而銀翹散及金銀花可以增加LPS誘發分泌IL-10。由銀翹散的HPLC成份分析中，找到銀翹散指標成分綠原酸、木犀草素、牛蒡素及橙皮素，其中最大量的成份為綠原酸，我們也證實綠原酸會增加LPS誘發RAW 264.7分泌IL-10；另外我們也發現橙皮素可抑制A549及THP-1的TNFα、IL-1β、IL-6、IL

-8的分泌，同時結果也顯示橙皮素可抑制發炎細胞的附著作用及ICAM-1與VCAM-1的表現。我們進一步發現銀翹散橙皮素可減少NF-κB與AP-1對其控制序列結合作用，並且橙皮素可阻斷IκB 及MAPK 路徑。因此，銀翹散有抑制LPS造成的肺部發炎作用及免疫調節作用，有助於急性肺部發炎的治療。